Author

A. Henke

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 1 Organism:mouse· 1

Frequent Co-Authors

B. Löffler· 2 shared C. Ehrhardt· 2 shared C. Häring· 2 shared S. Deinhardt-Emmer· 2 shared S. Nietzsche· 2 shared A. S. Mosig· 1 shared C. Brandt· 1 shared C. K. Glass· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersMicrobiology

B. Löffler

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

SARS-CoV-2 causes severe alveolar inflammation and barrier dysfunction

S. Deinhardt-Emmer, S. Böttcher, C. Häring, L. Giebeler, A. Henke, R. Zell, F. Hornung, C. Brandt, M. Marquet, A. S. Mosig, M. W. Pletz, M. Schacke, J. Roedel, R. Heller, S. Nietzsche, , C. Ehrhardt

This study shows SARS-CoV-2 infects alveolar epithelial cells, causing inflammation and barrier dysfunction, while endothelial cells are damaged through cytokine release from infected epithelial cells.

bioRxiv · Aug 31, 2020Microbiology

Early postmortem mapping of SARS-CoV-2 RNA in patients with COVID-19 and correlation to tissue damage

S. Deinhardt-Emmer, D. Wittschieber, J. Sanft, S. Kleemann, S. Elschner, K. F. Haupt, V. Vau, C. Häring, J. Rödel, A. Henke, C. Ehrhardt, M. Bauer, M. Philipp, N. Gassler, S. Nietzsche, , G. Mall

The paper maps SARS-CoV-2 RNA distribution in 11 deceased COVID-19 patients and correlates it with tissue damage and systemic inflammation.

bioRxiv · Jul 1, 2020Immunology

Cell-specific discrimination of desmosterol and desmosterol mimetics confers selective regulation of LXR and SREBP pathways in macrophages

E. D. Muse, S. Yu, C. R. Edillor, J. Tao, N. J. Spann, T. D. Troutman, J. S. Seidman, A. Henke, J. T. Roland, K. A. Ozeki, J. G. McDonald, J. Bahadorani, S. Tsimikas, T. R. Grossman, M. S. Tremblay, C. K. Glass

This study shows that desmosterol and its mimetics selectively activate LXR in macrophages without inducing SREBP1c-mediated hypertriglyceridemia, unlike conventional LXR agonists.

bioRxiv · Feb 11, 2018Cell Biology