Author

M. Ostermaier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

P. Beli· 2 shared A. Barros· 1 shared A. Bradley· 1 shared A. Bufe· 1 shared A. Ciprianidis· 1 shared A. de Jaime-Soguero· 1 shared A. Garcia del Arco· 1 shared B. Fu· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCell Biology

P. Beli

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Author profile →

2 papers

Recent·Most cited

Wnt signaling recruits KIF2A to the spindle to ensure chromosome congression and alignment during mitosis

A. Bufe, A. Garcia del Arco, M.-I. Hennecke, M. Ostermaier, A. de Jaime-Soguero, Y.-C. Lin, A. Ciprianidis, U. Engel, P. Beli, H. Bastians, S. P. Acebron

This paper identifies Wnt signaling's role in mitosis by recruiting KIF2A to the spindle, ensuring chromosome congression and alignment in somatic and pluripotent cells.

bioRxiv · Dec 22, 2020Cell Biology

ATM orchestrates the DNA-damage response to counter toxic non-homologous end-joining at broken replication forks

G. Balmus, D. Pilger, J. Coates, M. Demir, M. Sczaniecka-Clift, A. Barros, M. Woods, B. Fu, F. Yang, E. Chen, M. Ostermaier, T. Stankovic, H. Ponstingl, M. Herzog, K. Yusa, , S. P. P. Jackson

The paper identifies how ATM counteracts toxic non-homologous end-joining at broken replication forks by showing that loss of NHEJ components or BRCA1-A complex confers resistance to DNA-damaging agents in ATM-deficient cells.

bioRxiv · May 24, 2018Cell Biology