Author

m. ozkan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. Solanki· 2 shared J. E. G. Medellin· 2 shared A. T. Alpar· 1 shared B. A. Naved· 1 shared J. A. Hubbell· 1 shared J. Hubbell· 1 shared J. Wertheim· 1 shared K. M. Koss· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

A. Solanki

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2 papers

Recent·Most cited

Myofibroblast differentiation is governed by adhesion mechanics, and inhibition of the stress sensor Talin2 reverses lung fibrosis

M. J. V. WHITE, M. Ozkan, J. E. G. Medellin, A. Solanki, J. Hubbell

The paper identifies Talin2 as a key regulator of myofibroblast differentiation and shows that its inhibition reverses fibrosis in lung and kidney models.

bioRxiv · Jun 7, 2021 · PreprintImmunology

Blocking antibodies against integrin-a3, integrin-aM, and integrin-aMb2 de-differentiate myofibroblasts and reverse lung and kidney fibroses in a mouse model

M. WHITE, m. ozkan, J. E. G. Medellin, M. Raczy, K. M. Koss, A. Solanki, Z. J. Zhang, A. T. Alpar, B. A. Naved, J. Wertheim, J. A. Hubbell

Blocking antibodies against integrins-3, -M, and -Mβ2 reverse lung and kidney fibrosis in a mouse model by de-differentiating myofibroblasts.

bioRxiv · Jun 7, 2021 · PreprintImmunology