Author

Miyah R. Davis

Cedars-Sinai Medical Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

Altan RentsendorjiD· 3 shared Dieu-Trang S. Fuchs· 3 shared Keith L. BlackiD· 3 shared Maya Koronyo‐HamaouiiD· 3 shared Yosef Koronyo· 3 shared Alexander V. LjubimoviD· 2 shared Alfredo A. Sadun· 2 shared Bhakta Prasad GaireiD· 2 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

Altan RentsendorjORCID

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Author profile →

3 papers

Recent·Most cited

Synergistic retinal UCHL1 dysregulation and synaptic vulnerability reflect Alzheimer's disease severity

Altan Rentsendorj, Jean‐Philippe Vit, A. Hutton, Yosef Koronyo, Saba Shahin, Edward Robinson, Ernesto Barrón, Anthony Rodriguez, Bhakta Prasad Gaire, Ousman Jallow, Julia Sheyn, Miyah R. Davis, Alexander V. Ljubimov, Stuart L. Graham, Vivek K. Gupta, , Maya Koronyo‐Hamaoui

The paper identifies UCHL1 dysregulation and synaptic loss in the retina as markers of Alzheimer's disease severity using integrated multi-omics analysis.

bioRxiv · Feb 5, 2026 · PreprintNeuroscience

Retinal ganglion cell vulnerability to pathogenic tau in Alzheimer's disease

Miyah R. Davis, Edward Robinson, Yosef Koronyo, Elena Salobrar‐García, Altan Rentsendorj, Bhakta Prasad Gaire, Nazanin Mirzaei, Rakez Kayed, Alfredo A. Sadun, Alexander V. Ljubimov, Lon S. S. Schneider, Debra Hawes, Keith L. Black, Dieu-Trang S. Fuchs, Maya Koronyo‐Hamaoui

This study shows that pathological tau isoforms in retinal ganglion cells (RGCs) are linked to RGC degeneration in Alzheimer's disease, with significant correlations between tau levels and disease severity.

Acta Neuropathologica Communications · Feb 15, 2025Peer-reviewedPathology

Epigenetic derepression of H3K9me3 mitigates Alzheimer-related pathology and improves cognition via immunomodulation and Vgf induction

Dieu-Trang S. Fuchs, Jean‐Philippe Vit, Altan Rentsendorj, Julia Sheyn, Haoshen Shi, Yosef Koronyo, Oksana M. Chepurna, Miyah R. Davis, Jered W. Wilson, Lon S. S. Schneider, Stuart L. Graham, Vivek Gupta, Margaret L. Fahnestock, Tao Sun, Michael T. Kleinman, , Maya Koronyo‐Hamaoui

This study shows that inhibiting H3K9me3 with ETP69 reduces AD pathology and improves cognition by modulating neuroinflammation and inducing Vgf.

bioRxiv · Oct 15, 2024 · PreprintNeuroscience