Author

N. Bhutani

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1 Research Focus:development & morphogenesis· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Baldessari· 1 shared A. Christodoulou· 1 shared A. Colville· 1 shared A. E. Vo· 1 shared A. Gretch· 1 shared A. Li· 1 shared B. M. Del Rosario· 1 shared C. English· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperSystems Biology

A. Baldessari

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A single-cell transcriptomic atlas of the pigtail macaque lung

T. Kim, J. A. Cornelius, V. Sindhu, T. Duell, S. Sun, S. C. Chima, H. Huang, O. Cervantes, R. P. Kapur, H. Zhao, B. M. Del Rosario, N. Bhutani, E. Fettig, M. Waldorf, D. Bajaj, , K. M. Adams Waldorf

This paper constructs a single-cell transcriptomic atlas of the pigtail macaque lung, revealing high similarity to human lung cell types and providing a reference for studying respiratory diseases in a translational model.

bioRxiv · Nov 9, 2026 · PreprintSystems Biology

Transient non-integrative nuclear reprogramming promotes multifaceted reversal of aging in human cells

T. J. Sarkar, M. Quarta, S. Mukherjee, A. Colville, P. Paine, L. Doan, C. M. Tran, C. R. Chu, S. Horvath, N. Bhutani, T. A. Rando, V. Sebastiano

This study shows that transient non-integrative nuclear reprogramming reverses aging in human cells by rejuvenating epigenetic clocks and cellular functions without losing identity.

bioRxiv · Mar 18, 2019Cell Biology