Author

N. E. Garelis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

J. S. Cox· 2 shared A. W. Roberts· 1 shared A. Zilinskas· 1 shared B. Hill· 1 shared B. Luckie· 1 shared D. Jiminez-Morales· 1 shared D. Swaney· 1 shared G. Barton· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

J. S. Cox

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2 papers

Recent·Most cited

Cas9+ conditionally immortalized neutrophil progenitors as a tool for genome wide CRISPR screening for neutrophil differentiation and function

R. M. Jong, K. Ching, N. E. Garelis, A. Zilinskas, X. Nguyenla, S. Rawal, B. Hill, B. Luckie, L. Shallow, J. S. Cox, G. Barton, S. Stanley

This paper introduces a Cas9+ER-Hoxb8 neutrophil progenitor cell line for CRISPR screening to study neutrophil differentiation and function, identifying key genes like Cebpe and the WASH complex.

bioRxiv · Jul 19, 2022Immunology

Global proteomic profiling of primary macrophages during M. tuberculosis infection identifies TAX1BP1 as a mediator of autophagy targeting

J. M. Budzik, N. E. Garelis, T. Repasy, A. W. Roberts, L. Popov, T. J. Parry, D. Jiminez-Morales, D. Swaney, J. R. Johnson, N. J. Krogan, J. S. Cox

The paper uses proteomic analysis to identify TAX1BP1 as a mediator of autophagy targeting M. tuberculosis during macrophage infection.

bioRxiv · Jan 31, 2019 · PreprintMicrobiology