Author

J. M. Budzik

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:proteomics· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

J. S. Cox· 3 shared D. Jimenez-Morales· 2 shared J. R. Johnson· 2 shared N. J. Krogan· 2 shared T. Repasy· 2 shared A. W. Roberts· 1 shared B. S. Zha· 1 shared B. W. Newton· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersMicrobiology

J. S. Cox

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Mycobacterium tuberculosis resides in lysosome-poor monocyte-derived lung cells during chronic infection

W. Zheng, I.-C. Chang, J. Limberis, J. M. Budzik, B. S. Zha, Z. Howard, L. Chen, J. D. Ernst

This study identifies lysosome-poor monocyte-derived lung cells as a niche for Mtb persistence during chronic infection, suggesting a target for host-directed therapy.

bioRxiv · Jan 19, 2023Immunology

Ceragenins and antimicrobial peptides kill bacteria through distinct mechanisms

G. Mitchell, M. R. Silvis, K. C. Talkington, J. M. Budzik, C. E. Dodd, J. M. Paluba, E. A. Oki, K. L. Trotta, D. J. Licht, D. Jimenez-Morales, S. Chou, P. B. Savage, C. A. Gross, M. A. Marletta, J. S. Cox

This study compares the mechanisms of action of cationic antimicrobial peptides and ceragenins, finding distinct bacterial responses despite some overlapping effects.

bioRxiv · Oct 20, 2020Microbiology

Comparative analysis of macrophage post-translational modifications during intracellular bacterial pathogen infection

J. R. Johnson, T. Parry, T. Repasy, K. M. Geiger, E. Verschueren, J. M. Budzik, D. Jimenez-Morales, B. W. Newton, E. Powell, L. Coscoy, D. A. Portnoy, N. J. Krogan, J. S. Cox

This study uses proteomics to analyze post-translational modifications in macrophages infected with three intracellular pathogens, revealing pathogen-specific molecular responses and a novel mechanism of virus-bacterial synergy.

bioRxiv · May 27, 2020 · PreprintMicrobiology

Global proteomic profiling of primary macrophages during M. tuberculosis infection identifies TAX1BP1 as a mediator of autophagy targeting

J. M. Budzik, N. E. Garelis, T. Repasy, A. W. Roberts, L. Popov, T. J. Parry, D. Jiminez-Morales, D. Swaney, J. R. Johnson, N. J. Krogan, J. S. Cox

The paper uses proteomic analysis to identify TAX1BP1 as a mediator of autophagy targeting M. tuberculosis during macrophage infection.

bioRxiv · Jan 31, 2019 · PreprintMicrobiology