Author

N. G. Barra

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 4

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell signaling & trafficking· 3 Research Focus:host-pathogen & immune response· 3 Research Focus:metabolism & bioenergetics· 3 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

B. M. Duggan· 3 shared J. D. Schertzer· 3 shared A. K. Tamrakar· 2 shared B. D. Henriksbo· 2 shared F. F. Anhe· 2 shared J. F. Cavallari· 2 shared K. P. Foley· 2 shared D. M. Sloboda· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersPhysiology

B. M. Duggan

Mutual co-authors in cluster

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4 papers

Recent·Most cited

Gut microbiota-based vaccination engages innate immunity to improve blood glucose control in obese mice

B. M. Duggan, A. K. Tamrakar, N. G. Barra, F. F. Anhe, G. Paniccia, J. G. Wallace, H. D. Stacey, M. S. Miller, D. M. Sloboda, J. D. Schertzer

This study shows that gut microbiota-based vaccination improves blood glucose control in obese mice by engaging innate immunity, requiring bacterial components and specific immune proteins.

bioRxiv · Jul 15, 2021Physiology

Metabolic endotoxemia is dictated by the type of lipopolysaccharide

F. F. Anhe, N. G. Barra, J. F. Cavallari, B. D. Henriksbo, J. Schertzer

The paper shows that the type of lipopolysaccharide (LPS) determines metabolic endotoxemia effects, with E. coli LPS causing dysglycemia and inflammation while R. sphaeroides LPS has beneficial effects.

bioRxiv · Jul 13, 2021Physiology

Inflammation promotes adipocyte lipolysis via IRE1 kinase

K. P. Foley, Y. Chen, N. G. Barra, M. Heal, K. Kwok, A. K. Tamrakar, W. Chi, B. M. Duggan, B. D. Henriksbo, Y. Liu, J. D. Schertzer

This study identifies IRE1 kinase as a mediator of inflammation-induced adipocyte lipolysis, distinct from adrenergic or hormonal regulation.

bioRxiv · Apr 7, 2020Physiology

RIPK2 dictates insulin responses to tyrosine kinase inhibitors in obese mice

B. M. Duggan, J. F. Cavallari, K. P. Foley, N. G. Barra, J. D. Schertzer

This study shows that RIPK2 limits the insulin sensitizing effect of gefitinib, while imatinib increases insulin secretion in obese mice, indicating TKI actions on RIPK2 dictate divergent insulin responses.

bioRxiv · Apr 3, 2020Physiology