Author

N. Hirata

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Fujii· 1 shared A. Wiberg· 1 shared C. Nakashima· 1 shared D. Furniss· 1 shared G. L. Lukacs· 1 shared H. Takahashi· 1 shared H. Tateishi· 1 shared I. Hamano· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. Fujii

Mutual co-authors in cluster

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2 papers

Recent·Most cited

HERC3 E3 ligase provides an ERAD branch eliminating select membrane proteins

Y. Kamada, Y. Ohnishi, C. Nakashima, A. Fujii, M. Terakawa, I. Hamano, U. Nakayamada, S. Katoh, N. Hirata, H. Tateishi, R. Fukuda, H. Takahashi, G. L. Lukacs, T. Okiyoneda

This study identifies HERC3 as a cytosolic E3 ligase that facilitates ERAD of specific polytopic membrane proteins like CFTR, distinct from RNF5/185.

bioRxiv · Oct 16, 2023 · PreprintCell Biology

An SNP variant MT1-MMP with a defect in its collagenolytic activity confers the fibrotic phenotype of Dupuytren's Disease

Y. Itoh, M. Ng, A. Wiberg, K. Inoue, N. Hirata, K. B. Paiva, N. Ito, K. Dzobo, N. Sato, V. Gifford, Y. Fijita, M. Inada, D. Furniss

This study identifies an SNP variant in MMP14 that reduces collagenolytic activity, contributing to the fibrotic phenotype of Dupuytren's Disease.

bioRxiv · Jun 9, 2020Biochemistry