Author

N. Ramakrishnan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:ChIP-seq· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:Bayesian inference· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

S. R. B. Pillai· 2 shared A. J. Minn· 1 shared A. N. Soni· 1 shared B. N. B. Prabhu· 1 shared I. Asangani· 1 shared J. Benci· 1 shared J. Chen· 1 shared L. Tian· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersBioengineering

S. R. B. Pillai

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3 papers

Recent·Most cited

Antagonistic histone post-translational modifications improve the fidelity of epigenetic inheritance - a Bayesian perspective

B. N. B. Prabhu, A. N. Soni, S. R. B. Pillai, N. Ramakrishnan

The paper models how antagonistic histone PTMs improve epigenetic inheritance fidelity using Bayesian methods and simulations.

bioRxiv · May 7, 2024 · PreprintBioengineering

High fidelity epigenetic inheritance: Information theoretic model predicts k-threshold filling of histone modifications post replication

N. Ramakrishnan, S. R. B. Pillai, R. Padinhateeri

The paper develops an information-theoretic model to predict how histone modifications are inherited post-replication, showing that enzymes may use a k-threshold filling algorithm to recover parental modifications.

bioRxiv · May 25, 2021Biophysics

Cell cycle checkpoints cooperate to suppress DNA and RNA associated molecular pattern recognition and anti-tumor immune responses

R. A. Greenberg, J. Chen, S. M. Harding, N. Ramakrishnan, L. Tian, J. Benci, W. Li, A. J. Minn, I. Asangani

The paper shows that disrupting cell cycle checkpoints can restore DNA damage-induced inflammation and anti-tumor immune responses in cancer cells.

bioRxiv · Jun 24, 2020Cell Biology