Author

N. Rodrigues Mantuano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Zippelius· 2 shared A. Boersch· 1 shared A. Carlos Fonseca· 1 shared A. Filardy· 1 shared A. R. Todeschini· 1 shared A. Zingg· 1 shared C. de Souza Bastos· 1 shared D. Lardinois· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

A. Zippelius

Mutual co-authors in cluster

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintJun 29, 2023

Engagement of sialylated glycans with Siglec receptors on myeloid suppressor cells inhibit anti-cancer immunity via CCL2

R. Wieboldt, E. Carlini, L. Chia-wei…·N. Rodrigues Mantuano

This study shows that sialylated glycans on myeloid suppressor cells interact with Siglec receptors to suppress anti-cancer immunity through CCL2-mediated mechanisms.

DOI 10.1101/2023.06.29.547025

mouseproteomics

𝕏 PostBluesky

Cancer BiologybioRxivNov 5, 2019

Hyperglycemia enhances cancer immune evasion by inducing alternative macrophage polarization through increased O-GlcNAcylation

N. Rodrigues Mantuano, M. A. Stanczak, I. de Araujo Oliveira…·H. Laubli

This study shows hyperglycemia promotes cancer immune evasion by increasing O-GlcNAcylation in tumor-associated macrophages, shifting their polarization to a pro-tumorigenic M2-like phenotype.

DOI 10.1158/2326-6066.cir-19-0904

humanproteomics

𝕏 PostBluesky