Author

P. C. Cook

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

A. S. MacDonald· 2 shared E. L. Houlder· 2 shared D. Conn· 1 shared D. Thomson· 1 shared F. R. Svedberg· 1 shared G. D. Brown· 1 shared G. Howell· 1 shared G. Vere· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersImmunology

A. S. MacDonald

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2 papers

Recent·Most cited

Aspergillus-mediated allergic airway inflammation is triggered by dendritic cell recognition of a defined spore morphotype, a process that can be targeted via antifungal therapeutics.

E. L. Houlder, S. Gago, G. Vere, D. Conn, S. Khan, D. Thomson, M. Shepherd, R. Lebedinec, G. D. Brown, M. Bromley, A. S. MacDonald, P. C. Cook

This study identifies a specific stage of Aspergillus fumigatus spore development that activates dendritic cells to trigger allergic airway inflammation and shows antifungal drugs can reduce this process.

Journal of Allergy and Clinical Immunology · Mar 1, 2025Peer-reviewedImmunology

Mgl2+ cDC2s coordinate fungal allergic airway type 2, but not type 17, inflammation.

P. C. Cook, S. L. Brown, E. L. Houlder, S. Baker, F. R. Svedberg, G. Howell, M. Bertuzzi, L. Boon, J. E. Konkel, J. E. Allen, A. S. MacDonald

The paper identifies Mgl2+ cDC2s as key drivers of type 2, but not type 17, fungal allergic airway inflammation in mice.

bioRxiv · Nov 24, 2023Immunology