Author

P. Kamat

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:human· 2 Assay:imaging· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Approach:experimental assay· 1 Approach:machine learning (classical)· 1

Frequent Co-Authors

A. Agrawal· 2 shared B. Starich· 2 shared C. Min· 2 shared J. M. Phillip· 2 shared J. Walston· 2 shared P.-H. Wu· 2 shared A. Giri· 1 shared A. J. Crawford· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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2 shared papersBioengineering

A. Agrawal

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Author profile →

3 papers

Recent·Most cited

Single-cell morphology encodes functional subtypes of senescence in aging human dermal fibroblasts

P. Kamat, N. Macaluso, C. Min, Y. Li, A. Agrawal, A. Winston, L. Pan, B. Starich, T. Stewert, P.-H. Wu, J. Fan, J. Walston, J. M. Phillip

This study identifies functional senescence subtypes in aging human dermal fibroblasts using single-cell morphology and machine learning, showing subtype-specific responses to senotherapies.

bioRxiv · May 10, 2024Bioengineering

Profiling Dynamic Patterns of Single-cell Motility

D. Maity, N. Sivakumar, P. Kamat, N. Zamponi, C. Min, W. Du, H. Jayatilaka, A. Giri, A. Johnston, B. Starich, M. Karl, A. Agrawal, D. Riley, L. Venturitti, A. Melnick, , J. M. Phillip

This study introduces CaMI, a framework to classify single-cell motility patterns, revealing heterogeneity and dynamics not captured by traditional ensemble methods.

bioRxiv · Sep 21, 2022Bioengineering

E-cadherin interacts with EGFR resulting in hyper-activation of ERK in multiple models of breast cancer

G. C. Russo, A. J. Crawford, D. J. Clark, J. Cui, R. Carney, M. N. Karl, B. Su, B. A. Starich, T.-S. Lih, P. Kamat, Q. Zhang, P.-h. Wu, M.-H. Lee, H. S. Leong, V. W. Rebecca, , D. Wirtz

This study shows E-cadherin interacts with EGFR to hyper-activate ERK, promoting proliferation in breast cancer models and identifying a potential therapeutic target.

bioRxiv · Nov 4, 2020Cancer Biology