Author

P. L. Moura

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Organism:human· 3 Assay:proteomics· 3 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. M. Toye· 2 shared A. D'Alessandro· 1 shared A. J. Mead· 1 shared A.-C. Bjorklund· 1 shared C. E. Severn· 1 shared C. Martins Freire· 1 shared C. Ziegenhain· 1 shared D. C. Granados· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersCell Biology

A. M. Toye

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Author profile →

3 papers

Recent·Most cited

Complete absence of GLUT1 does not impair human terminal erythroid differentiation

C. Martins Freire, N. R. King, M. Dzieciatkowska, D. Stephenson, P. L. Moura, J. G. G. Dobbe, G. J. Streekstra, A. D'Alessandro, A. M. Toye, T. J. Satchwell

This study shows that complete absence of GLUT1 in human erythroid cells does not impair differentiation or membrane properties, but leads to metabolic changes and increased oxidative stress.

bioRxiv · Jan 10, 2024 · PreprintCell Biology

Erythroid differentiation intensifies RNA mis-splicing in SF3B1-mutant myelodysplastic syndromes with ring sideroblasts

P. L. Moura, T. Mortera-Blanco, I. J. F. Hofman, G. Todisco, W. W. Kretzschmar, A.-C. Bjorklund, M. Creignou, M. Hagemann-Jensen, C. Ziegenhain, D. C. Granados, I. Barbosa, G. Walldin, M. Jansson, N. Ashley, A. J. Mead, , E. Hellstrom-Lindberg

This study investigates SF3B1-mutant myelodysplastic syndromes with ring sideroblasts by isolating RS and analyzing their development and mis-splicing during erythroid differentiation.

bioRxiv · Apr 11, 2023Cancer Biology

Characterizing the polarization continuum of macrophage subtypes M1, M2a and M2c

T. C. Oates, P. L. Moura, S. J. Cross, K. Roberts, H. E. Baum, K. Haydn-Smith, M. C. Wilson, K. J. Heesom, C. E. Severn, A. M. Toye

This study characterizes four macrophage subtypes using proteomics and functional assays, showing M2c macrophages upregulate pro-inflammatory markers and dexamethasone induces partial repolarization from M1 to M2c.

bioRxiv · Jun 13, 2022Immunology