Author

P. S. Hume

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:flow cytometry· 1 Assay:imaging· 1

Frequent Co-Authors

A. Campos Codo· 1 shared A. Getahun· 1 shared A. van Bokhoven· 1 shared B. Crute· 1 shared B. Pena· 1 shared C. Hauer· 1 shared C. M. Magin· 1 shared D. T. Merrick· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

A. Campos Codo

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Author profile →

2 papers

Recent·Most cited

Tissue-engineered models of lung cancer premalignancy

R. Blomberg, K. Sompel, C. Hauer, B. Pena, J. Driscoll, P. S. Hume, D. T. Merrick, M. A. Tennis, C. M. Magin

This paper develops a tissue-engineered model of human lung cancer premalignancy using hydrogel-embedded lung slices exposed to cigarette smoke carcinogens, showing the formation of premalignant lesions and testing chemoprevention agents.

bioRxiv · Mar 15, 2023 · PreprintBioengineering

Polymorphic KIR3DL3 expression modulates tissue-resident and innate-like T cells.

W. H. Palmer, L. A. Leaton, A. Campos Codo, P. S. Hume, B. Crute, M. Stone, A. van Bokhoven, R. P. Tobin, M. McCarter, W. J. Janssen, J. Roest, S. Zhu, J. Petersen, J. P. Vivian, J. Rossjohn, , P. J. Norman

This study identifies KIR3DL3 expression in T cells, showing it is upregulated by unconventional stimulation and varies by individual genetic variants.

bioRxiv · Aug 17, 2022Immunology