Author

R. E. White

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Biophysics· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

V. M. Tan· 2 shared A. Day· 1 shared A. Szymula· 1 shared B. J. Yachnin· 1 shared B. S. Holder· 1 shared C. A. S. A. Minetti· 1 shared D. P. Remeta· 1 shared H. D. Nguyen· 1 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersCancer Biology

V. M. Tan

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Epstein-Barr virus protein EBNA-LP engages YY1 through leucine-rich motifs to promote naïve B cell transformation

J. M. Cable, R. E. White, N. M. Reinoso-Vizcaino, M. A. Luftig

This study identifies EBNA-LP's role in promoting naive B cell transformation by interacting with YY1 through conserved leucine-rich motifs.

bioRxiv · Jan 7, 2024Microbiology

Saracatinib synergizes with enzalutamide to downregulate androgen receptor activity in castration resistant prostate cancer

R. E. White, M. Bannister, A. Day, H. E. Bergom, V. M. Tan, J. Hwang, H. D. Nguyen, J. Drake

This study shows that combining saracatinib with enzalutamide reduces androgen receptor activity and DNA replication in CRPC cell lines, leading to increased apoptosis.

bioRxiv · Apr 22, 2023 · PreprintCancer Biology

Massively parallel, computationally-guided design of a pro-enzyme

B. J. Yachnin, L. R. Azouz, R. E. White, C. A. S. A. Minetti, D. P. Remeta, V. M. Tan, J. M. Drake, S. D. Khare

This paper designs pro-enzymes using computational methods and validates their activity regulation through protease cleavage.

bioRxiv · Mar 25, 2021Biophysics

Epstein-Barr virus nuclear antigen EBNA-LP is essential for transforming naive B cells, and facilitates recruitment of transcription factors to the viral genome.

A. Szymula, R. D. Palermo, I. J. Groves, M. Ba abdullah, B. S. Holder, R. E. White

This paper shows EBNA-LP is essential for transforming naive B cells and facilitates transcription factor recruitment to the viral genome, with distinct effects on different B cell subsets.

bioRxiv · Aug 22, 2017Microbiology