Author

R. Lingerak

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Assay:fluorescence / live-cell microscopy· 1 Organism:mouse· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. W. Smith· 2 shared B. Wang· 2 shared M. Buck· 2 shared X. Shi· 2 shared C. Cuizon· 1 shared C. Herting· 1 shared D. Hambardzumyan· 1 shared D. Nikolov· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersBiophysics

A. W. Smith

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2 papers

Recent·Most cited

The SAM Domain of EphA2 Inhibits Ligand-Independent Clustering and Activation

X. Shi, R. Lingerak, P. Shrestha, M. Buck, B. Wang, A. W. Smith

This study investigates the role of the SAM domain in EphA2, showing it inhibits ligand-independent clustering and kinase activity in both full-length and intracellular domain constructs.

bioRxiv · Oct 5, 2023 · PreprintBiophysics

Cell Surface Multimeric Assemblies Regulate Canonical and Noncanonical EphA2 Receptor Tyrosine Kinase Signaling

X. Shi, R. Lingerak, C. Herting, Y. Ge, S. Kim, P. Toth, C. Cuizon, J. Zheng, L. H. Chao, K. Sossey-Alaoui, M. Buck, S. Singh, V. Varadan, J. Himanen, D. Hambardzumyan, , B. Wang

This study identifies molecular interfaces that mediate EphA2 receptor assembly and distinguishes between canonical and noncanonical signaling pathways.

bioRxiv · Apr 11, 2021 · PreprintBiophysics