Author

R. M. Fu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

J. Hu· 2 shared V. L. Dao Thi· 2 shared A. Bryan· 1 shared A. McKnight· 1 shared A. Prawira· 1 shared B. Qu· 1 shared B. W. Ogunkolade· 1 shared C. E. Jones· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersMicrobiology

J. Hu

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A high-content RNA-based imaging assay reveals integrin beta 1 as a cofactor for cell entry of non-enveloped hepatitis E virus

R. M. Fu, Z. Engels, J. A. Wheis, J. Muerle, M. Kloehn, D. Todt, J. Hu, E. Steinmann, T. Boettler, P.-Y. Lozach, S. M. Lemon, V. L. Dao Thi

This study identifies integrin beta 1 as a cofactor for naked hepatitis E virus entry, revealing distinct endocytic pathways for naked and enveloped HEV particles.

bioRxiv · Oct 27, 2023 · PreprintMicrobiology

Human pluripotent stem cell-derived hepatocyte-like cells for hepatitis D virus studies

H. Chi, B. Qu, A. Prawira, L. Maurer, J. Hu, R. M. Fu, F. A. Lempp, Z. Zhang, D. Grimm, X. Wu, S. Urban, V. L. Dao Thi

This study shows that hepatocyte-like cells derived from human pluripotent stem cells are permissive to hepatitis D virus infection and can be used to study HDV biology and test therapies.

bioRxiv · Oct 13, 2023 · PreprintMicrobiology

HIV-1 accessory protein Vpr interacts with REAF and mitigates its associated antiviral 1 activity.

J. M. Gibbons, K. M. Marno, R. Pike, W.-y. J. Lee, C. E. Jones, B. W. Ogunkolade, C. Pardieu, A. Bryan, R. M. Fu, G. Warnes, P. A. Rowley, R. D. Sloan, A. McKnight

This paper shows that HIV-1 Vpr interacts with REAF, a macrophage-expressed antiviral factor, and promotes its degradation to enhance HIV replication.

bioRxiv · Oct 30, 2019Microbiology