Author

S. E. Henrickson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. C. Villani· 1 shared A. Veillette· 1 shared C. G. Pfeifer· 1 shared C.-J. Lu· 1 shared E. Hajaj· 1 shared E. J. Wherry· 1 shared F. Fenninger· 1 shared G. Eisenberg· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

A. C. Villani

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2 papers

Recent·Most cited

Mutation of an L-Type Calcium Channel Gene Leads to a Novel Human Primary Cellular Immunodeficiency

F. Fenninger, S. R. Stanwood, C.-J. Lu, C. G. Pfeifer, O. Khan, N. D. Romberg, R. Herati, E. J. Wherry, K. C. O'Boyle, S. E. Henrickson, K. Maurer, M. A. Ruffner, W. A. Jefferies, K. E. Sullivan

This study identifies a mutation in the Cav1.4 gene causing a primary immunodeficiency with immune dysfunction and recurrent infections.

bioRxiv · Dec 4, 2019 · PreprintImmunology

SLAMF6 deficiency augments tumor killing and skews towards an effector phenotype revealing it as a new T cell checkpoint

E. Hajaj, G. Eisenberg, S. Klein, S. Frankenburg, S. Merims, I. Ben David, T. Eisenhaure, S. E. Henrickson, A. C. Villani, N. Hacohen, N. Abudi, R. Abramovich, J. E. Cohen, T. Peretz, A. Veillette, M. Lotem

This study shows that SLAMF6 deficiency enhances T cell tumor-killing ability and shifts T cells toward an effector phenotype, suggesting SLAMF6 as a new immune checkpoint.

bioRxiv · Oct 31, 2019Physiology