Author

S. F. Monaghan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

A. Ayala· 1 shared A. M. Fredericks· 1 shared A. M. Jamieson· 1 shared A. R. D. Jordon· 1 shared B. M. Hall· 1 shared C. T. Lefort· 1 shared F. Maschietto· 1 shared G. J. Nau· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. Ayala

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Identification and Mechanistic Basis of non-ACE2 Blocking Neutralizing Antibodies from COVID-19 Patients with Deep RNA Sequencing and Molecular Dynamics Simulations

A. M. Fredericks, K. W. East, Y. Shi, J. Liu, F. Maschietto, A. Ayala, W. G. Cioffi, M. Cohen, W. G. Fairbrother, C. T. Lefort, G. J. Nau, M. M. Levy, J. Wang, V. S. Batista, G. P. Lisi, S. F. Monaghan

The paper identifies non-ACE2 blocking neutralizing antibodies in COVID-19 patients using deep RNA sequencing and molecular dynamics simulations, showing their potential for personalized therapy.

bioRxiv · Jun 29, 2022Immunology

Pulmonary infection interrupts acute cutaneous wound healing through disruption of chemokine signals

M. J. Crane, Y. Xu, S. F. Monaghan, B. M. Hall, J. E. Albina, W. L. Henry, H. L. Tran, K. R. P. Chhabria, A. R. D. Jordon, L. Carlsen, A. M. Jamieson

This study shows that pulmonary infection impairs cutaneous wound healing by reducing chemokine signaling, which can be reversed by restoring chemokine levels.

bioRxiv · May 8, 2020 · PreprintImmunology