Author

S. Favre

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1 Assay:metagenomics· 1 Organism:mouse· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

B. Pillet· 2 shared D. Kressler· 2 shared A. Mendez-Godoy· 1 shared C. Vionnet· 1 shared D. Siva Sankar· 1 shared D. Zehn· 1 shared G. M. Fimia· 1 shared G. Murat· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

B. Pillet

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The ULK1 effector BAG2 regulates autophagy initiation by modulating AMBRA1 localization

J. Dengjel, D. Siva Sankar, S. Kaeser-Pebernard, C. Vionnet, S. Favre, L. Oliveira De Marchioro, B. Pillet, J. Zhou, W. J. Kovacs, D. Kressler, M. Antonioli, G. M. Fimia

This study identifies BAG2 as an effector of the ULK1 complex that modulates AMBRA1 localization to regulate autophagy initiation under different nutritional conditions.

bioRxiv · Dec 8, 2023Cell Biology

Dedicated chaperones coordinate co-translational regulation of ribosomal protein production with ribosome assembly to preserve proteostasis

B. Pillet, A. Mendez-Godoy, G. Murat, S. Favre, M. Stumpe, L. Falquet, D. Kressler

The paper shows that dedicated chaperones prevent degradation of RPL3 and RPL4 mRNAs in yeast by coordinating ribosomal protein production with ribosome assembly.

bioRxiv · Oct 5, 2021Cell Biology

Attenuation of chronic antiviral T cell responses through constitutive COX2-dependent prostanoid synthesis by lymph node fibroblasts

K. Schaeuble, H. Cannelle, S. Favre, H.-Y. Huang, S. Oberle, D. Zehn, S. A. Luther

The paper identifies lymph node fibroblastic reticular cells (FRC) as a source of COX2-dependent PGE2 that suppresses T cell responses during chronic infection.

bioRxiv · Oct 30, 2018Immunology