Author

S. Hurst

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:metabolism & bioenergetics· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

T. Betz· 2 shared A. E. M. Beedle· 1 shared A. Elosegui-Artola· 1 shared A.-L. Le Roux· 1 shared B. E. Vos· 1 shared B. Falcones· 1 shared D. Sturgill· 1 shared G. Hager· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersBiophysics

T. Betz

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The force loading rate drives cell mechanosensing through both reinforcement and fluidization

I. Andreu, B. Falcones, S. Hurst, N. Chahare, X. Quiroga, A.-L. Le Roux, Z. Kechagia, A. E. M. Beedle, A. Elosegui-Artola, X. Trepat, R. Farre, T. Betz, I. Almendros, P. Roca-Cusachs

This study shows that the rate of force application drives cell mechanosensing through reinforcement and fluidization mechanisms.

bioRxiv · Mar 8, 2021Biophysics

Intracellular softening and fluidification reveals a mechanical switch of cytoskeletal material contributions during division

S. Hurst, B. E. Vos, T. Betz

The paper shows that during cell division, the cytoplasm softens and fluidifies, with microtubules and actin switching roles in controlling mechanical properties.

bioRxiv · Jan 7, 2021 · PreprintBiophysics

Activation of the Crtc2/Creb1 transcriptional network in skeletal muscle enhances weight loss during intermittent fasting

N. Bruno, J. C. Nwachukwu, S. Srinivasan, D. Sturgill, G. Hager, S. Hurst, S.-S. Sheu, M. Conkright, K. W. Nettles

This study shows that Crtc2/Creb1 overexpression in skeletal muscle enhances mitochondrial function and promotes weight loss during intermittent fasting by improving metabolic flux and reversing fasting-induced mitochondrial downregulation.

bioRxiv · Jun 27, 2020 · PreprintPhysiology