Author

S. M. Henson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. N. Akbar· 2 shared E. C. Carroll· 2 shared J. Schroth· 2 shared L. A. Callender· 2 shared A. Appios· 1 shared A. Kelly· 1 shared A. Vallejo· 1 shared C. L. Bennett· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersImmunology

A. N. Akbar

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Convergent evolution of monocyte differentiation in adult skin instructs Langerhans cell identity

A. Appios, J. Davies, S. Sirvent, S. Henderson, S. Trzebanski, J. Schroth, M. L. Law, I. Boal Carvalho, H. Yuan-Hao Kan, S. Lovlekar, C. Major, A. Vallejo, N. J. Hall, M. Ardern-Jones, S. M. Henson, , C. L. Bennett

The paper investigates how monocyte differentiation in adult skin leads to Langerhans cell identity through intrinsic and extrinsic factors.

bioRxiv · Nov 13, 2023Immunology

GATA3 controls mitochondrial biogenesis in primary human CD4+ T cells during DNA damage

L. A. Callender, J. Schroth, E. C. Carroll, L. E. L. Romano, E. Hendy, A. Kelly, P. Lavender, A. N. Akbar, J. P. Chapple, S. M. Henson

This study identifies a novel mechanism by which GATA3 regulates mitochondrial biogenesis in primary human CD4+ T cells in response to DNA damage.

bioRxiv · May 15, 2020 · PreprintImmunology

Mitochondrial mass governs the extent of T cell senescence

L. A. Callender, E. C. Carroll, E. A. Bober, A. N. Akbar, E. Solito, S. M. Henson

This study shows that mitochondrial mass differences between CD4+ and CD8+ T cells drive their distinct susceptibility to senescence, with reduced mitochondrial function inducing a CD8+ -like phenotype in CD4+ T cells.

bioRxiv · May 4, 2019Immunology