Author

S. M. Mutchler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Organism:rat· 1

Frequent Co-Authors

A. L. Marciszyn· 1 shared A. Porter· 1 shared A. R. Subramanya· 1 shared A. Vandevender· 1 shared C. J. Baty· 1 shared H. E. Vorndran· 1 shared J. L. Brodsky· 1 shared L. M. Hendershot· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. L. Marciszyn

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2 papers

Recent·Most cited

PhysiologyJournal of the American Society of NephrologyPeer-reviewedJul 10, 2024

Mineralocorticoid receptor-independent activation of ENaC in bile duct ligated mice

X.-P. Wang, S. M. Mutchler, R. Carrisoza-Gaytan…·O. B. Kashlan

This study shows that bile acid elevation in liver disease activates ENaC and causes fluid retention independently of RAAS, offering new therapeutic targets.

DOI 10.1681/asn.0000000000000442

mouserat

Cell BiologybioRxivPreprintJan 13, 2024

Excess dietary sodium partially restores salt and water homeostasis caused by loss of the endoplasmic reticulum molecular chaperone, GRP170, in the mouse nephron

A. Porter, H. E. Vorndran, A. L. Marciszyn…·T. M. Buck

This study shows that excess dietary sodium partially restores electrolyte imbalance and kidney injury markers in GRP170-deficient mice but not weight or tubule integrity.

DOI 10.1101/2024.01.13.575426

mouseexperimental assay