Author

A. Vandevender

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:metabolism & bioenergetics· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1 Organism:rat· 1

Frequent Co-Authors

B. Kaufman· 1 shared B. McMahon· 1 shared B. Mushala· 1 shared C. J. Baty· 1 shared I. Scott· 1 shared J. Manning· 1 shared L. M. Satlin· 1 shared M. Al-bataineh· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

B. Kaufman

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

TCA Cycle Dysfunction and Amino Acid Catabolism Drive Hepatic Steatosis in Mice with HFpEF

B. Mushala, M. Stoner, J. Manning, P. Bugga, N. Bhattarai, M. Sanjani, B. McMahon, A. Vandevender, S. Mullett, B. Kaufman, S. Shiva, S. Gelhaus, M. Jurczak, I. Scott

This study shows that TCA cycle dysfunction and amino acid catabolism in mice with HFpEF drive hepatic steatosis similar to human NAFLD.

bioRxiv · Jun 3, 2024 · PreprintPhysiology

Mineralocorticoid receptor-independent activation of ENaC in bile duct ligated mice

X.-P. Wang, S. M. Mutchler, R. Carrisoza-Gaytan, M. Al-bataineh, C. J. Baty, A. Vandevender, P. Srinivasan, R. J. Tan, M. J. Jurczak, L. M. Satlin, O. B. Kashlan

This study shows that bile acid elevation in liver disease activates ENaC and causes fluid retention independently of RAAS, offering new therapeutic targets.

bioRxiv · Sep 19, 2023Physiology