Author

S. Mijailovich

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Assay:proteomics· 2 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Organism:rat· 1

Frequent Co-Authors

A. Adhikari· 1 shared A. Combs· 1 shared C. A. Johnson· 1 shared C. D. Vera· 1 shared C. L. del Rio· 1 shared C. Zambataro· 1 shared H. Gong· 1 shared J. A. Spudich· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperBiophysics

A. Adhikari

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2 papers

Recent·Most cited

Myosin in autoinhibited off state(s), stabilized by mavacamten, can be recruited via inotropic effectors

W. Ma, C. L. del Rio, L. Qi, M. Prodanovic, S. Mijailovich, C. Zambataro, H. Gong, R. Shimkunas, S. Gollapudi, S. Nag, T. Irving

This paper shows that mavacamten stabilizes myosin in off states, which can still be recruited by physiological inotropic effectors to maintain cardiac function.

bioRxiv · Apr 10, 2023 · PreprintPhysiology

Myosin motor domains carrying mutations implicated in early or late onset Hypertrophic Cardiomyopathy have similar properties

C. D. Vera, C. A. Johnson, J. Walklate, A. Adhikari, M. Svicevic, S. Mijailovich, A. Combs, S. J. Langer, K. M. Ruppel, J. A. Spudich, M. A. Geeves, L. A. LEINWAND

The paper analyzes myosin motor domain mutations in Hypertrophic Cardiomyopathy to compare early and late onset mutations, finding no clear functional differences but identifying distinct changes from DCM mutations.

bioRxiv · Aug 10, 2019Biophysics