Author

S. R. Cuddy

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. R. Cliffe· 3 shared C. Boutell· 2 shared J. B. Suzich· 2 shared S. Dochnal· 2 shared A. Babnis· 1 shared A. K. Francois· 1 shared A. L. Whitford· 1 shared A. R. Schinlever· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersMicrobiology

A. R. Cliffe

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Author profile →

3 papers

Recent·Most cited

c-Jun Signaling During Initial HSV-1 Infection Modulates Latency to Enhance Later Reactivation in addition to Directly Promoting the Progression to Full Reactivation

S. A. Dochnal, A. L. Whitford, A. K. Francois, P. A. Krakowiak, S. R. Cuddy, A. R. Cliffe

This study shows c-Jun signaling during HSV-1 initial infection promotes latency and enhances later reactivation by facilitating the transition from Phase I to Phase II reactivation.

Journal of Virology · Feb 20, 2024Peer-reviewedMicrobiology

PML-Dependent Memory of Type I Interferon Treatment Results in a Restricted Form of HSV Latency

J. B. Suzich, S. R. Cuddy, H. Baidas, S. Dochnal, E. Ke, A. Babnis, C. Boutell, A. R. Cliffe

This study shows that type I interferon treatment during HSV-1 infection leads to restricted latency by altering viral genome positioning through PML-NBs.

bioRxiv · Feb 3, 2021Immunology

Neuronal Hyperexcitability is a DLK-dependent Trigger of HSV-1 Reactivation that can be Induced by IL-1

S. R. Cuddy, A. R. Schinlever, S. Dochnal, J. B. Suzich, P. Kundu, T. K. Downs, M. Farah, B. Desai, C. Boutell, A. R. Cliffe

This study shows that neuronal hyperexcitability, induced by IL-1β, triggers HSV-1 reactivation through DLK/JNK pathway and histone phosphorylation.

bioRxiv · Apr 16, 2020 · PreprintMicrobiology