Author

Sabine Karolus

German Cancer Research Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Angelika Wörner· 2 shared Cindy KörneriD· 2 shared Janina MülleriD· 2 shared Luisa E. SchwarzmülleriD· 2 shared Sara Burmester· 2 shared Stefan WiemanniD· 2 shared Almut SchulzeiD· 1 shared Birgitta E. MichelsiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

Angelika Wörner

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

PAK1 activation drives divergent resistance mechanisms to aromatase inhibition and Tamoxifen in a luminal A breast cancer model

Luisa E. Schwarzmüller, Janina Müller, Efstathios–Iason Vlachavas, Lukas Beumers, Lena Fleischhacker, Sara Burmester, Angelika Wörner, Sabine Karolus, Dominic Helm, Cindy Körner, Stefan Wiemann

This study shows PAK1 activation leads to different resistance mechanisms against Tamoxifen and aromatase inhibitors in luminal A breast cancer by affecting apoptosis and proliferation through distinct pathways.

Molecular Oncology · Jun 5, 2026Peer-reviewedCancer Biology

GLYATL1 is associated with metabolic and epigenetic changes and with endocrine resistance in luminal breast cancer

Janina Müller, Emre Sofyalı, Luisa E. Schwarzmüller, Yael Aylon, Eviatar Weizman, Lisa Schlicker, Katherine Kelly, Simone Borgoni, Simin Oz, Cole Stocker, Sara Burmester, Angelika Wörner, Sabine Karolus, Birgitta E. Michels, Daniela Heiss, , Stefan Wiemann

This study identifies GLYATL1 as a mediator of endocrine resistance in luminal breast cancer by promoting metabolic and epigenetic changes that counteract estrogen deprivation.

Clinical Epigenetics · Apr 29, 2026Peer-reviewedCancer Biology