Janina Müller

The paper quantifies fitness costs of protein overexpression in E. coli, showing growth collapse due to membrane proteome displacement, not translocation pathway saturation.

This study shows PAK1 activation leads to different resistance mechanisms against Tamoxifen and aromatase inhibitors in luminal A breast cancer by affecting apoptosis and proliferation through distinct pathways.

This study identifies GLYATL1 as a mediator of endocrine resistance in luminal breast cancer by promoting metabolic and epigenetic changes that counteract estrogen deprivation.

This paper introduces PHORCE, a method to quantify phage amplification rates and reveals wide variation in phage-antibiotic interactions across Escherichia coli phages.