Author

Sarah J. Morse

University of California, Davis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:gene regulatory network inference· 1 Assay:GWAS summary statistics· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Alex S. NordiD· 3 shared César P. CanalesiD· 2 shared Diana QuinteroiD· 2 shared Iva ZdilariD· 2 shared Jason T. LambertiD· 2 shared Linda Su-FeheriD· 2 shared Tracy L. WarreniD· 2 shared Tyler W. StradleighiD· 2 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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3 shared papersNeuroscience

Alex S. NordORCID

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Author profile →

3 papers

Recent·Most cited

Association of neurotransmitter pathway polygenic risk with specific symptom profiles in psychosis

Tracy L. Warren, Justin D. Tubbs, Tyler A. Lesh, Mylena B. Corona, Sarvenaz Pakzad, Marina Albuquerque, Praveena Singh, Vanessa C. Zarubin, Sarah J. Morse, Pak Chung Sham, Cameron S. Carter, Alex S. Nord

This study links neurotransmitter pathway polygenic risk to specific symptom profiles in psychosis, identifying associations between glutamate and dopamine pathways with cognitive control and global functioning.

Molecular Psychiatry · Mar 15, 2024Peer-reviewedGenetic & Genomic Medicine

Parallel functional testing identifies enhancers active in early postnatal mouse brain

Jason T. Lambert, Linda Su-Feher, Karol Cichewicz, Tracy L. Warren, Iva Zdilar, Yurong Wang, Kenneth J. Lim, Jessica Haigh, Sarah J. Morse, César P. Canales, Tyler W. Stradleigh, Erika Castillo Palacios, Viktoria Haghani, Spencer D Moss, Hannah Parolini, , Alex S. Nord

The paper identifies and validates enhancers active in early postnatal mouse brain using a modified MPRA approach and rAAV delivery.

eLife · Oct 4, 2021Peer-reviewedNeuroscience

Deletion of a non-canonical regulatory sequence causes loss of Scn1a expression and epileptic phenotypes in mice

Jess Haigh, Anna Adhikari, Nycole A. Copping, Tyler W. Stradleigh, A. Ayanna Wade, Rinaldo Catta-Preta, Linda Su-Feher, Iva Zdilar, Sarah J. Morse, Timothy A. Fenton, Anh Nguyen, Diana Quintero, Samrawit Agezew, Michael Sramek, Ellie J. Kreun, , Alex S. Nord

This study shows that deleting a non-canonical regulatory region of Scn1a in mice leads to reduced Scn1a expression and epileptic symptoms.

Genome Medicine · Apr 26, 2021Peer-reviewedNeuroscience