Author

Sarah Woelfle

Universität Ulm

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

Francesco RoselliiD· 3 shared Tobias M. BoeckersiD· 3 shared Albert Christian LudolphiD· 2 shared Dhruva DeshpandeiD· 2 shared Florian olde HeuveliD· 2 shared Jens MichaelisiD· 2 shared Michael SchöniD· 2 shared Alberto CataneseiD· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersNeuroscience

Francesco RoselliORCID

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3 papers

Recent·Most cited

CLARITY increases sensitivity and specificity of fluorescence immunostaining in long-term archived human brain tissue

Sarah Woelfle, Dhruva Deshpande, Simone Feldengut, Heiko Braak, Kelly Del Tredici, Francesco Roselli, Karl A. Deisseroth, Jens Michaelis, Tobias M. Boeckers, Michael Schön

The paper introduces hCLARITY, a clearing method that improves the sensitivity and specificity of fluorescence immunostaining in archived human brain tissue for high-resolution imaging.

BMC Biology · May 23, 2023Peer-reviewedNeuroscience

Interleukin-13 and its receptor are synaptic proteins involved in plasticity and neuroprotection

Shun Li, Florian olde Heuvel, Rida Rehman, Zhenghui Li, Oumayma Aousji, Albrecht Fröhlich, Wanhong Zhang, Alison Conquest, Sarah Woelfle, Michael Schoen, Caitlin Claire O'Meara, Richard Lee Reinhardt, David Voehringer, Jan Kassubek, Albert Christian Ludolph, , Francesco Roselli

The paper identifies IL-13 and its receptor as neuronal synaptic proteins that enhance synaptic activity and neuroprotection in response to injury.

bioRxiv · Dec 1, 2021 · PreprintNeuroscience

Shank2 expression identifies a subpopulation of glycinergic interneurons involved in nociception and altered in an autism mouse model

Florian olde Heuvel, Najwa Ouali Alami, Hanna Wilhelm, Dhruva Deshpande, Elmira Khatamsaz, Alberto Catanese, Sarah Woelfle, Michael Schön, Sanjay Jain, Stefanie Grabrucker, Albert Christian Ludolph, Chiara Verpelli, Jens Michaelis, Tobias M. Boeckers, Francesco Roselli

The paper identifies a subpopulation of glycinergic interneurons in the spinal cord regulated by Shank2, whose dysfunction contributes to pain hypersensitivity in ASD mouse models.

bioRxiv · May 24, 2020 · PreprintNeuroscience