Author

Stefan Paulusch

University of Bonn

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Elena De DomenicoiD· 3 shared Alessio RicciiD· 1 shared Andreas SchlitzeriD· 1 shared Andrew M. BelliD· 1 shared Anika ReifschneideriD· 1 shared Anja CapelliD· 1 shared Arthur LiesziD· 1 shared Astrid FeiteniD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersNeuroscience

Elena De DomenicoORCID

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3 papers

Recent·Most cited

Deep FLASH-seq profiling of purified canine sensory neurons uncovers species-specific signatures relevant to pain and itch

Paula Ledesma Fernández, Brandi Butler, Heidi Theis, Stefan Paulusch, Elena De Domenico, Greg A. Weir, Andrew M. Bell

The paper profiles canine sensory neurons using FLASH-seq to identify species-specific molecular signatures relevant to pain and itch, revealing both conserved and divergent features compared to human and mouse.

Pain · Sep 2, 2026Peer-reviewedNeuroscience

TREM2 expression level is critical for microglial state, metabolic capacity and efficacy of TREM2 agonism

Astrid Feiten, Kilian Dahm, Kai Schlepckow, Bettina van Lengerich, Jung H. Suh, Anika Reifschneider, Benedikt Wefers, Laura M. Bartos, Karin Wind-Mark, Lis de Weerd, T Ulas, Elena De Domenico, Pia Grundschöttel, Stefan Paulusch, Benjamin Tast, , Christian Haass

This study investigates how TREM2 expression levels affect microglial function, metabolism, and response to TREM2 agonists in a mouse model of Alzheimer's disease.

Nature Communications · Jan 24, 2026Peer-reviewedNeuroscience

Dysregulation of gene expression during gastrulation results in impaired primitive erythropoiesis and vascular development in Trim71-KO embryos

Tobias Beckröge, Bettina Jux, Hannah Seifert, Hannah Theobald, Elena De Domenico, Stefan Paulusch, Marc Beyer, Andreas Schlitzer, Elvira Mass, Waldemar Kolanus

This study shows that Trim71 deficiency during gastrulation disrupts gene expression, leading to defects in erythropoiesis and vascular development, causing embryonic lethality.

bioRxiv · Jul 12, 2024 · PreprintDevelopmental Biology