Author

T. Schneiders

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:E. coli· 2 Organism:bacteria (other)· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. F. Cunningham· 1 shared A. Meynert· 1 shared B. Waclaw· 1 shared C. Collins· 1 shared C. Icke· 1 shared D. Scales· 1 shared E. C. Goodall· 1 shared I. R. Henderson· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperMicrobiology

A. F. Cunningham

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Author profile →

3 papers

Recent·Most cited

Transposon mutagenesis screen in Klebsiella pneumoniae identifies genetic determinants required for growth in human urine and serum

J. Gray, V. V. L. Torres, E. C. Goodall, S. A. McKeand, D. Scales, C. Collins, L. Wetherall, Z. J. Lian, J. A. Bryant, M. T. Milner, K. Dunne, C. Icke, J. Rooke, T. Schneiders, P. A. Lund, , I. R. Henderson

This study identifies essential genes for Klebsiella pneumoniae growth in human urine and serum using transposon mutagenesis, highlighting potential antimicrobial targets.

bioRxiv · May 31, 2023Microbiology

Rapid Evolution of Colistin Resistance in a Bioreactor Model of Infection of Klebsiella pneumoniae

J.-C. J. Castellanos, B. Waclaw, A. Meynert, T. Schneiders

This study shows that colistin resistance in K. pneumoniae evolves rapidly through spontaneous mutations in regulatory genes during treatment.

bioRxiv · Oct 3, 2022Microbiology

The Envelope Proteome Changes Driven By RamA Overproduction in Klebsiella pneumoniae That Enhance Acquired β-Lactam Resistance

J.-C. Jimenez-Castellanos, W. A. K. Wan Nur Ismah, Y. Takebayashi, J. Findlay, T. Schneiders, K. J. Heesom, M. B. Avison

This study shows RamA overproduction in K. pneumoniae enhances β-lactam resistance by increasing efflux pumps and reducing OmpK35 porin, without altering β-lactamase levels.

bioRxiv · May 3, 2017Microbiology