Author

Thomas H. Sisson

University of Michigan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Genetics· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

Alexey I. NesvizhskiiiD· 1 shared Ammara Q Virk· 1 shared Daniel A. LawrenceiD· 1 shared Daniel TorrenteiD· 1 shared David S. Ginsberg· 1 shared Duane E. Day· 1 shared Dudley K. StricklandiD· 1 shared Jeffrey Craig HorowitziD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPathology

Alexey I. NesvizhskiiORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

PAI-1 Interaction with Sortilin Related Receptor-1 is Required for Lung Fibrosis

Thomas H. Sisson, John J. Osterholzer, Lisa Leung, Venkatesha Basrur, Alexey I. Nesvizhskii, Natalya Subbotina, Mark Warnock, Daniel Torrente, Ammara Q Virk, Sergey S Gutor, Jeffrey Craig Horowitz, Mary M. Migliorini, Dudley K. Strickland, Kevin K. Kim, Steven Kenneth Huang, Daniel A. Lawrence

The paper identifies SorlA as a novel PAI-1 interactor required for lung fibrosis, showing SorlA deficiency protects against fibrosis in mice and SorlA is upregulated in human IPF lung tissue.

JCI Insight · Apr 29, 2025Peer-reviewedPathology

Optimizing CRISPR/Cas9 System to Precisely Model Plasminogen Activator Inhibitor-1 Point Mutations in Mice

Yang Liu, Thomas L. Saunders, Thomas H. Sisson, Robert Andrew Blackburn, David S. Ginsberg, Duane E. Day

This study optimizes CRISPR/Cas9 to model specific PAI-1 mutations in mice, finding that mutation distance from the DSB site affects editing efficiency.

bioRxiv · Jan 26, 2018 · PreprintGenetics