Author

Tibor Kovács

Eötvös Loránd University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Cell Biology· 1 Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 3 Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1

Frequent Co-Authors

Tibor VellaiiD· 2 shared Andor Vizi· 1 shared András RubicsiD· 1 shared Anikó NagyiD· 1 shared Attila BodaiD· 1 shared Balázs HangyaiD· 1 shared Bálint KirályiD· 1 shared Diána BalázsfiiD· 1 shared

Activity by Year

2020-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersNeuroscience

Tibor VellaiORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Huntington's disease-associated ankyrin repeat palmitoyl transferases are rate-limiting factors in lysosome formation and fusion

Győző Szenci, Attila Boda, Anikó Nagy, Dorottya Károlyi, András Rubics, Zsombor Szőke, Gergő Falcsik, Tibor Kovács, Péter Lőrincz, Gábor Juhász, Szabolcs Takáts

This study identifies Hip14 and Patsas as rate-limiting factors in lysosome formation and fusion, linking defective palmitoylation to Huntington's disease-like symptoms in Drosophila.

PLoS Genetics · Dec 31, 2025Peer-reviewedCell Biology

Altered reactivity to threatening stimuli in Drosophila models of Parkinson's disease, revealed by a trial-based assay

Márton Kajtor, Viktor Billes, Bálint Király, Patricia Karkusova, Tibor Kovács, Hannah Stabb, Katalin Sviatkó, Andor Vizi, Eszter Ujvári, Diána Balázsfi, Sophie E Seidenbecher, Duda Kvitsiani, Tibor Vellai, Balázs Hangya

This study uses a trial-based assay to show that Drosophila with Parkinson's-related genetic mutations exhibit altered reactivity to threatening stimuli, revealing potential insights into disease mechanisms.

eLife · Jul 10, 2025Peer-reviewedNeuroscience

Rab2 is a potent new target for enhancing autophagy in the treatment of Parkinson's disease

Janka Szinyákovics, Eszter Kiss, Fanni Keresztes, Tibor Vellai, Tibor Kovács

This paper identifies Rab2 as a target for enhancing autophagy in Parkinson's disease models, showing improved neuronal function and lifespan.

bioRxiv · Aug 30, 2020 · PreprintGenetics