Torri D. Wilson

CGRP receptor-expressing neurons in the central amygdala contributes to injury-induced pain hypersensitivity

This study identifies CGRPR-expressing neurons in the central amygdala as key players in pain hypersensitivity, showing their role in both injury-induced and spontaneous pain responses.

The parabrachial to central amygdala circuit is a key mediator of injury-induced pain sensitization

This study shows the PbN[->]CeA circuit contributes to injury-induced pain hypersensitivity but not baseline nociception in mice.

Bidirectional modulation of pain-related behaviors in the zona incerta

This study identifies an inhibitory neural circuit from CeA-PKCδ neurons to the zona incerta that bidirectionally modulates pain-related behaviors in mice.

4R-Tobacco-Cembranoid is a Positive Allosteric Modulator of the Alpha7 Nicotinic Acetylcholine Receptor that modulates inflammation-induced thermal hypersensitivity in mice

This study identifies 4R-Tobacco-Cembranoid as a positive allosteric modulator of 7nAChRs that reduces thermal hypersensitivity and inflammation in mice, with sex-specific effects.

Sex differences in pain-related behaviors and clinical progression of disease in mouse models of visceral pain

This study examines sex differences in visceral pain behaviors and disease progression in mouse models of acute and persistent colon inflammation, finding sex-specific responses and disease outcomes.

Cell-type specificity of neuronal excitability and morphology in the central amygdala

This study identifies cell-type-specific differences in excitability and morphology between PKCδ+ and Som+ neurons in the central amygdala and links these differences to behavioral changes in neuropathic pain.