Anisha P. Adke

The paper develops a 3D agent-based model of the bilateral central amygdala to predict pain outcomes by integrating neuronal data from CGRPR and PKCδ cells.

This study investigates VGF's role in nucleus accumbens synaptic and opioid-evoked plasticity, finding it modulates drug-evoked adaptations and behavior.

This study shows the PbN[->]CeA circuit contributes to injury-induced pain hypersensitivity but not baseline nociception in mice.

This study identifies an inhibitory neural circuit from CeA-PKCδ neurons to the zona incerta that bidirectionally modulates pain-related behaviors in mice.

This study identifies cell-type-specific differences in excitability and morphology between PKCδ+ and Som+ neurons in the central amygdala and links these differences to behavioral changes in neuropathic pain.

The paper identifies Tjp1b as an intracellular scaffold required for electrical synapse formation and shows its asymmetric localization and function at the postsynaptic side.