Author

V. Sanchorawala

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

C. V. Edwards· 2 shared G. J. Murphy· 2 shared R. M. Giadone· 2 shared S. Ghosh· 2 shared A. Presser· 1 shared A. Staron· 1 shared C. Fraser· 1 shared C. S. Golden· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

C. V. Edwards

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Abnormal global longitudinal strain and reduced serum inflammatory markers in cardiac AL amyloidosis patients without significant amyloid fibril deposition

C. V. Edwards, G. M. Ferri, J. Villegas-Galaviz, S. Ghosh, P. Singh Bawa, F. Wang, E. Klimtchuk, T. B. Ajayi, G. J. Morgan, T. Prokaeva, A. Staron, F. L. Ruberg, V. Sanchorawala, R. M. Giadone, G. J. Murphy

The paper identifies early molecular and functional signatures in cardiac AL amyloidosis patients with abnormal GLS and reduced inflammatory markers despite no significant amyloid fibril deposition.

bioRxiv · Mar 14, 2024Cell Biology

Mapping Cellular Response to Destabilized Transthyretin Reveals Cell- and Amyloidogenic Protein-Specific Signatures

S. Ghosh, C. Villacorta-Martin, J. Lindstrom-Vautrin, D. Kenney, C. S. Golden, C. V. Edwards, V. Sanchorawala, L. H. Connors, R. M. Giadone, G. J. Murphy

The paper exposes cells to destabilized TTR variants and analyzes transcriptional and epigenetic responses to identify cell- and protein-specific signatures of amyloidogenic stress.

bioRxiv · Aug 17, 2022Cell Biology

Clonal plasma cells in AL amyloidosis are dependent on pro survival BCL-2 family proteins and sensitive to BH3 mimetics

C. Fraser, A. Presser, V. Sanchorawala, S. Sarosiek, K. Sarosiek

This study identifies that clonal plasma cells in AL amyloidosis depend on BCL-2 family proteins and are sensitive to BH3 mimetics, suggesting potential therapeutic strategies.

bioRxiv · Feb 6, 2019 · PreprintCancer Biology