Author

V. Vitvitsky

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:metabolism & bioenergetics· 2 Assay:metabolomics· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1

Frequent Co-Authors

R. Banerjee· 3 shared C. A. Lyssiotis· 2 shared R. Kumar· 2 shared A. Andren· 1 shared A. Guha· 1 shared A. J. Finch· 1 shared A. P. Landry· 1 shared A. Tailleux· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersBiochemistry

R. Banerjee

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Author profile →

3 papers

Recent·Most cited

Sulfide oxidation promotes hypoxic angiogenesis and neovascularization

R. Kumar, V. Vitvitsky, P. Seth, H. L. Hiraki, H. Bell, A. Andren, R. Singhal, B. Baker, C. A. Lyssiotis, Y. M. Shah, R. Banerjee

This study shows that sulfide oxidation via SQOR promotes hypoxic angiogenesis and neovascularization by reprogramming metabolic pathways in endothelial cells.

Nature Chemical Biology · Mar 20, 2024Peer-reviewedBiochemistry

A redox cycle with complex II promotes sulfide quinone oxidoreductase dependent H2S oxidation

R. Kumar, A. P. Landry, A. Guha, V. Vitvitsky, H.-J. Lee, K. Seike, P. Reddy, C. A. Lyssiotis, R. Banerjee

The paper identifies a redox cycle involving complex II and SQOR that sustains H2S oxidation when complex IV is inhibited, improving H2S clearance in cells exposed to microbiota-derived H2S.

bioRxiv · Sep 8, 2021 · PreprintBiochemistry

The hepatic compensatory response to elevated systemic sulfide promotes diabetes

R. N. Carter, M. T. G. Gibbins, M. E. Barrios-Llerena, S. E. Wilkie, P. L. Freddolino, M. Libiad, V. Vitvitsky, B. Emerson, T. Le Bihan, M. Brice, H. Su, S. G. Denham, N. Z. M. Homer, C. Mc Fadden, A. Tailleux, , N. M. Morton

This study shows that mice lacking the sulfide oxidation pathway enzyme TST exhibit metabolic abnormalities despite insulin sensitization, highlighting TST's role in hepatic metabolism and potential implications for sulfide-donor therapies.

bioRxiv · Apr 27, 2020Physiology