Author

Y. Samuels

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. A. Kolodziejczyk· 1 shared A. A. Schaffer· 1 shared A. Eisenberg-Lerner· 1 shared A. Javitt· 1 shared A. Kacen· 1 shared A. Satpathy· 1 shared A. Toste Rego· 1 shared A. Ulman· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperCancer Biology

A. A. Kolodziejczyk

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Temporal genomic analysis of melanoma rejection identifies regulators of tumor immune evasion

S. C. Shvefel, J. Pai, Y. Cao, L. Pal, R. Levy, W. Yao, K. Cheng, M. Zemanek, O. Bartok, C. Weller, Y. Yin, P. Du, E. Yakubovich, I. Orr, S. Ben-Dor, , Y. Samuels

The paper identifies Mif as a regulator of tumor immune evasion in low intra-tumor heterogeneity melanoma by analyzing single-cell RNA sequencing data and validating in patient data.

bioRxiv · Nov 29, 2023 · PreprintCancer Biology

The proteasome regulator PSME4 drives immune evasion and abrogates anti-tumor immunity in NSCLC

A. Javitt, M. D. Shmueli, M. P. Kramer, A. A. Kolodziejczyk, I. J. Cohen, I. Kamer, K. Litchfield, E. Bab-Dinitz, O. Zadok, V. Neiens, A. Ulman, L. Radomir, H. Wolf-Levy, A. Eisenberg-Lerner, A. Kacen, , Y. Merbl

This paper identifies PSME4 as a proteasome regulator that suppresses anti-tumor immunity in NSCLC by inhibiting immunoproteasome activity and antigen presentation.

bioRxiv · Oct 24, 2021 · PreprintCancer Biology

Cross-talk between the receptor tyrosine kinases AXL and ERBB3 regulates invadopodia formation in melanoma cells

O.-Y. Revach, O. Sandler, Y. Samuels, B. Geiger

This study identifies AXL and ERBB3 cross-talk as regulators of invadopodia formation in melanoma cells, showing AXL's dual role and ERBB3's compensatory activation upon AXL suppression.

bioRxiv · Sep 9, 2018Cancer Biology