Author

Yushi T. Redhead

King's College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Organism:mouse· 3 Assay:imaging· 2 Approach:statistical model· 2 Approach:experimental assay· 1

Frequent Co-Authors

Jeremy B. A. GreeniD· 3 shared Elizabeth FisheriD· 2 shared Victor L. J. TybulewicziD· 2 shared Anthony Christopher VernoniD· 1 shared Benedikt HallgrímssoniD· 1 shared Camilla SimmonsiD· 1 shared Christopher M. HenstridgeiD· 1 shared Diana CashiD· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersDevelopmental Biology

Jeremy B. A. GreenORCID

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3 papers

Recent·Most cited

Craniofacial dysmorphology in Down Syndrome is caused by increased dosage of Dyrk1a and at least three other genes

Yushi T. Redhead, Dorota Gibbins, Eva Lana‐Elola, Sheona Watson-Scales, Lisa Dobson, Matthias Krause, Karen J. Liu, Elizabeth Fisher, Jeremy B. A. Green, Victor L. J. Tybulewicz

The paper identifies four Hsa21-orthologous genes, including Dyrk1a, that cause craniofacial dysmorphology in Down syndrome mouse models by reducing neural crest cell proliferation.

bioRxiv · Jun 27, 2022 · PreprintDevelopmental Biology

MRI-guided histology of TDP-43 knock-in mice implicates parvalbumin interneuron loss, impaired neurogenesis and aberrant neurodevelopment in ALS-FTD

Ziqiang Lin, Eugene Kim, Mohi U. Ahmed, Gang Han, Camilla Simmons, Yushi T. Redhead, Jack Bartlett, Emiliano Peña‐Altamira, Isobel Callaghan, Matthew A. White, Nisha Singh, Stephen J. Sawiak, Tara L. Spires‐Jones, Anthony Christopher Vernon, Michael P. Coleman, , Jemeen Sreedharan

This study uses MRI and histology to identify structural and cellular changes in TDP-43 knock-in mice, linking them to ALS-FTD pathology and identifying PV+ interneuron loss as a potential biomarker.

Brain Communications · Apr 1, 2021Peer-reviewedNeuroscience

Application of high-resolution landmark-free morphometrics to a mouse model of Down Syndrome reveals a tightly localised cranial phenotype

Nicolas Toussaint, Yushi T. Redhead, Marta Vidal‐García, Lucas Lo Vercio, Wei Liu, Elizabeth Fisher, Benedikt Hallgrímsson, Victor L. J. Tybulewicz, Julia A. Schnabel, Jeremy B. A. Green

This paper applies a landmark-free morphometric method to a mouse model of Down syndrome, revealing localized cranial phenotypes not detectable with traditional landmark-based approaches.

Development · Mar 12, 2021Peer-reviewedGenetics