Author

Stela Virgilio

Department of Molecular and Cellular Biology, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, 14040-900, SP, Brazil;ORCID 0000-0001-5195-9965Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:gene regulatory network inference· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Jennifer Ann BlackiD· 2 shared Jeziel Dener DamascenoiD· 2 shared Luiz Ricardo Orsini TosiiD· 2 shared Richard McCullochiD· 2 shared Craig T. LapsleyiD· 1 shared Gabriel L. A. SilvaiD· 1 shared Matheus Silva e BastosiD· 1 shared Ricardo ObonagaiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 25 mutual co-author links

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2 shared papersMolecular Biology

Jennifer Ann BlackORCID

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2 papers

Recent·Most cited

Molecular BiologybioRxivPreprintNov 10, 2026

Replication-stress response shapes Leishmania genome plasticity through non-equivalent RAD9 and HUS1 functions.

Ricardo Obonaga, Jennifer Ann Black, Stela Virgilio…·Luiz Ricardo Orsini Tosi

This study shows that HUS1 and RAD9 subunits of the 9-1-1 checkpoint clamp have opposing roles in shaping genome plasticity in Leishmania major through distinct replication-stress responses and genome remodelling patterns.

DOI 10.64898/2026.11.08.765153

experimental assaychromatin & epigenetic regulation

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Molecular BiologybioRxivPreprintNov 4, 2024

Genome-wide mapping of RPA1 and RAD9 reveals the management of polycistronic transcription, replication initiation, and responses to replication stress in Leishmania

Jennifer Ann Black, Stela Virgílio, Matheus Silva e Bastos…·Luíz Ricardo Orsini Tosi

This study maps RPA1 and RAD9 in Leishmania to understand replication stress response, showing stress hotspots correlate with replication initiation and transcription termination.

1 citation

ChIP-seqstatistical model

𝕏 PostBluesky