Author

Matheus Silva e Bastos

Indiana University School of MedicineORCID 0000-0002-1834-6005Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Approach:statistical model· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Craig T. LapsleyiD· 1 shared Gabriel L. A. SilvaiD· 1 shared Jennifer Ann BlackiD· 1 shared Jeziel D. DamascenoiD· 1 shared Luíz Ricardo Orsini TosiiD· 1 shared Michael J. HolmesiD· 1 shared Richard McCullochiD· 1 shared Ronald C. WekiD· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMolecular Biology

Craig T. LapsleyORCID

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2 papers

Recent·Most cited

Cap-independent translation directs stress-induced differentiation of the protozoan parasite Toxoplasma gondii

Vishakha Dey, Michael J. Holmes, Matheus Silva e Bastos, Ronald C. Wek, William J. Sullivan

This study identifies cap-independent translation as a mechanism driving stress-induced differentiation in Toxoplasma gondii by regulating BFD1 protein synthesis.

Journal of Biological Chemistry · Nov 13, 2024Peer-reviewedMicrobiology

Genome-wide mapping of RPA1 and RAD9 reveals the management of polycistronic transcription, replication initiation, and responses to replication stress in Leishmania

Jennifer Ann Black, Stela Virgílio, Matheus Silva e Bastos, Gabriel L. A. Silva, Jeziel D. Damasceno, Craig T. Lapsley, Richard McCulloch, Luíz Ricardo Orsini Tosi

This study maps RPA1 and RAD9 in Leishmania to understand replication stress response, showing stress hotspots correlate with replication initiation and transcription termination.

bioRxiv · Nov 4, 2024 · PreprintMolecular Biology