Author

Ivan Menendez-Montes

Spanish National Centre for Cardiovascular ResearchORCID 0000-0001-5252-9502Unclaimed ORCID profile · follow to get notified of new preprints

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Developmental Biology· 2

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:metabolomics· 2 Organism:mouse· 2 Assay:proteomics· 2

Frequent Co-Authors

Alessia FerrariniiD· 2 shared Ana Vanessa Alonso· 2 shared Beatriz EscobariD· 2 shared Beatriz PalaciosiD· 2 shared Elena Bonzón‐KulichenkoiD· 2 shared Jesús VázqueziD· 2 shared Luis Jesús Jiménez‐BorregueroiD· 2 shared Manuel J. GómeziD· 2 shared

Activity by Year

2019-2020 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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2 shared papersDevelopmental Biology

Alessia FerrariniORCID

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2 papers

Recent·Most cited

Activation of amino acid metabolic program in response to impaired glycolysis in cardiac HIF1 deficient mice

Ivan Menendez-Montes, Beatriz Escobar, Manuel J. Gómez, Teresa Albendea-Gomez, Beatriz Palacios, Elena Bonzón‐Kulichenko, Ana Vanessa Alonso, Alessia Ferrarini, Luis Jesús Jiménez‐Borreguero, Jesús Vázquez, Silvia Martı́n-Puig

The paper investigates the effects of HIF1 deletion in cardiac progenitors, finding that it leads to impaired glycolysis and activation of an amino acid metabolic program without affecting heart development or function.

bioRxiv · May 23, 2020 · PreprintDevelopmental Biology

Metabolic reprogramming from glycolysis to amino acid utilization in cardiac HIF1α deficient mice.

Ivan Menendez-Montes, Beatriz Escobar, Beatriz Palacios, Manuel J. Gómez, Elena Bonzón‐Kulichenko, Alessia Ferrarini, Ana Vanessa Alonso, Luis Jesús Jiménez‐Borreguero, Jesús Vázquez, Silvia Martı́n-Puig

This study investigates the effects of HIF1α deletion in cardiac progenitors during heart development, finding that HIF1 is dispensable for cardiac growth and reveals metabolic reprogramming toward amino acid utilization.

bioRxiv · Mar 16, 2019 · PreprintDevelopmental Biology