Author

Rebecca Gilley

Babraham InstituteORCID 0000-0001-5315-0801Unclaimed ORCID profile · follow to get notified of new preprints

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Rebecca Gilley1 paper · Babraham Institute

Biology Disciplines

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Biochemistry· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

Glenn Robert MassoniD· 2 shared Graham W. NeilliD· 2 shared Simon J. CookiD· 2 shared Andrew M. KidgeriD· 1 shared Chao ZhangiD· 1 shared Dominic P. ByrneiD· 1 shared Eve Morrison· 1 shared Gideon E. BollagiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

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10 top collaborators · 39 mutual co-author links

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2 shared papersBiochemistry

Glenn Robert MassonORCID

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2 papers

Recent·Most cited

BiochemistryNature CommunicationsPeer-reviewedNov 17, 2025

RAF inhibitors activate the integrated stress response by direct activation of GCN2

Rebecca Gilley, Andrew M. Kidger, Graham W. Neill…·Simon J. Cook

This study shows RAF inhibitors activate the integrated stress response via direct GCN2 activation, independent of ERK1/2, leading to ATF4 and CHOP expression.

7 citations

humanproteomics

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BiochemistrybioRxivPreprintAug 14, 2024

Paradoxical Activation of GCN2 by ATP-competitive inhibitors via allosteric activation and autophosphorylation

Graham W. Neill, Vanesa Vinciauskaite, Marilyn Paul…·Glenn Robert Masson

The paper shows ATP-competitive inhibitors paradoxically activate GCN2 through allosteric activation and autophosphorylation, suggesting alternative strategies for GCN2 inhibition.

4 citations

proteomicsexperimental assay

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