Author

David R. Gibb

Cedars-Sinai Medical CenterORCID 0000-0001-5863-3239Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Assay:proteomics· 1

Frequent Co-Authors

Christian StehlikiD· 2 shared Arati Naveen KumariD· 1 shared Caroline Anne JefferiesiD· 1 shared Che‐Yu ChangiD· 1 shared Daniel J. WallaceiD· 1 shared Gabriela De Los Santos· 1 shared Gantsetseg TumurkhuuiD· 1 shared Graziela PerriiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersImmunology

Christian StehlikORCID

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2 papers

Recent·Most cited

Loss of TRIM21 drives UVB-induced systemic inflammation by regulating DNA-sensing pathways

Gantsetseg Tumurkhuu, Graziela Perri, Richard Moore, Lihong Huo, Arati Naveen Kumar, Richard I. Ainsworth, TRINITEE R. OLIVER, Gabriela De Los Santos, David R. Gibb, Jessica Carrière, Jeong Min Yu, Rachel Abuav, Lindsy J. Forbess, Daniel J. Wallace, MARIKO L. ISHIMORI, , Caroline Anne Jefferies

This study shows that loss of TRIM21 increases UVB-induced systemic inflammation by enhancing IFN expression through impaired STING signaling in mice.

Arthritis & Rheumatology · Jun 17, 2025Peer-reviewedImmunology

CDDO-Imidazole regulates RBC alloimmunization to the KEL antigen by activating Nrf2

Che‐Yu Chang, Rosario Hernández‐Armengol, Kausik Paul, Juneyoung Lee, Karina Nance, Tomohiro Shibata, Peibin Yue, Christian Stehlik, David R. Gibb

This study shows that CDDO-Im, an Nrf2 activator, reduces RBC alloimmunization to the KEL antigen by suppressing IFNβ-stimulated gene expression and promoting RBC recovery in mice.

bioRxiv · Apr 3, 2025 · PreprintImmunology