Author

Lionel A. J. Tintignac

University of BaselORCID 0000-0001-5902-1138Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Organism:human· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Michael SinnreichiD· 2 shared Alexander S. HamiD· 1 shared Alexander SchmidtiD· 1 shared Beat Erne· 1 shared Daniel J. HamiD· 1 shared Kathrin Chojnowska· 1 shared Markus A. RüeggiD· 1 shared Meric Ataman· 1 shared

Activity by Year

2019-2025 · peak 1/yr

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10 top collaborators · 25 mutual co-author links

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2 shared papersMolecular Biology

Michael SinnreichORCID

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2 papers

Recent·Most cited

Molecular BiologyScientific ReportsPeer-reviewedDec 21, 2025

Identification of new interactors of eIF3f by endogenous proximity-dependent biotin labelling in human muscle cells.

Lionel A. J. Tintignac, Nitish Mittal, Shahidul Alam…·Michael Sinnreich

The paper identifies new interactors of eIF3f in human muscle cells using BioID technology, revealing both nuclear and cytoplasmic interactions relevant to muscle structure and function.

DOI 10.1038/s41598-025-32702-7

humanproteomics

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Molecular BiologybioRxivPreprintAug 19, 2019

mTORC1 signaling is not essential for the maintenance of muscle mass and function in adult sedentary mice

Alexander S. Ham, Kathrin Chojnowska, Lionel A. J. Tintignac…·Markus A. Rüegg

This study shows that mTORC1 inhibition in adult mice does not cause muscle atrophy or functional decline, despite metabolic and morphological changes.

DOI 10.1101/738294

mouseproteomics

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