Author

Michael Sinnreich

University of BaselORCID 0000-0002-9835-1276Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Assay:DNA methylation· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1 Approach:statistical model· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Lionel A. J. TintignaciD· 2 shared Adeline Stiefvater· 1 shared Alexander S. HamiD· 1 shared Alexander SchmidtiD· 1 shared Beat Erne· 1 shared Daniel J. HamiD· 1 shared Jochen KinteriD· 1 shared Kathrin Chojnowska· 1 shared

Activity by Year

2019-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersMolecular Biology

Lionel A. J. TintignacORCID

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3 papers

Recent·Most cited

Identification of new interactors of eIF3f by endogenous proximity-dependent biotin labelling in human muscle cells.

Lionel A. J. Tintignac, Nitish Mittal, Shahidul Alam, Meric Ataman, Yusuf I. Ertuna, Thomas Bock, Beat Erne, Mihaela Zavolan, Michael Sinnreich

The paper identifies new interactors of eIF3f in human muscle cells using BioID technology, revealing both nuclear and cytoplasmic interactions relevant to muscle structure and function.

Scientific Reports · Dec 21, 2025Peer-reviewedMolecular Biology

ratioPCR delivers precise real-time quantification from multi-template PCR via mechanistic bias correction

Jochen Kinter, Adeline Stiefvater, Samira F Osterop, Michael Sinnreich

ratioPCR improves qPCR precision by correcting mechanistic biases, enabling accurate quantification of nucleic acid ratios in high-throughput applications.

bioRxiv · Nov 20, 2025 · PreprintMolecular Biology

mTORC1 signaling is not essential for the maintenance of muscle mass and function in adult sedentary mice

Alexander S. Ham, Kathrin Chojnowska, Lionel A. J. Tintignac, Shuo Lin, Alexander Schmidt, Daniel J. Ham, Michael Sinnreich, Markus A. Rüegg

This study shows that mTORC1 inhibition in adult mice does not cause muscle atrophy or functional decline, despite metabolic and morphological changes.

bioRxiv · Aug 19, 2019 · PreprintMolecular Biology