Guillaume Ferreira

This study shows that an obesogenic diet impairs social memory in mice by altering hippocampal CA2 excitability and oxytocin signaling, with specific effects on endocannabinoid plasticity and social novelty discrimination.

This study investigates how endocannabinoid-dopamine interactions in the hippocampus mediate incidental associations, showing that CB1 receptors in D1-receptor-expressing cells are critical for IA formation.

This study examines sex-dependent astrocytic morphology in the medial habenula and its modulation by metabolic and inflammatory factors.

This study shows that developmental vitamin A deficiency causes sex-specific changes in reward processing in adult mice by altering mesolimbic dopamine transmission.

This study shows that an obesogenic diet impairs memory consolidation in mice via the hippocampal endocannabinoid system and mTOR pathway, which can be reversed by CB1R blockade or mTOR inhibition.

This study shows that dopamine signaling in the medial prefrontal cortex encodes action value during reversal learning through D1/NMDA and D2/NMDA receptor heteromers.

This study shows that adjusting meal timing can reverse memory impairments and glucocorticoid signaling imbalances caused by constant access to high-calorie food in mice.

This study shows that time-restricted feeding improves memory and metabolic health in mice on an obesogenic diet by restoring thyroid hormone signaling in the hippocampus.

This study investigates the role of NAc D1 and D2 neurons in balancing feeding and exercise-mediated energy expenditure in mice, finding that manipulating these neurons affects energy balance and weight.

This study shows that an obesogenic diet during adolescence impairs specific memory types via overactivation of hippocampal pathways, and targeting these pathways can reverse deficits.

The paper shows lactate-dependent glycolysis shifts produce D-serine, linking astrocyte metabolism to synaptic and cognitive functions through CB1 receptor activation.

This study uses chemogenetic inhibition to show that periadolescent obesogenic diet affects hippocampus and amygdala memory functions differently, impairing hippocampus-dependent memory and enhancing amygdala-dependent memory.