Author

Terunaga Nakagawa

Vanderbilt UniversityORCID 0000-0001-6174-5012Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:database / resource· 1 Research Focus:differential expression· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Anirvan Ghosh· 1 shared Bianka K. Kohegyi· 1 shared Daniel B. McClatchyiD· 1 shared Danyang ZhangiD· 1 shared David Aaron JacobsoniD· 1 shared Edward H. KooiD· 1 shared Eliezer MasliahiD· 1 shared Ingo H. GregeriD· 1 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperNeuroscience

Anirvan Ghosh

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintSep 25, 2022

Modulatory mechanisms of TARP γ8-selective AMPA receptor therapeutics

Danyang Zhang, Remigijus Lapė, Saher Afshan Shaikh…·Ingo H. Greger

The paper investigates the modulatory mechanisms of TARP γ8-selective AMPA receptor therapeutics using cryo-EM structures, electrophysiology, and simulations, revealing ligand-dependent effects on receptor function.

1 citation

experimental assay

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NeuroscienceJournal of NeurosciencePeer-reviewedFeb 4, 2020

Neuronal L-Type Calcium Channel Signaling to the Nucleus Requires a Novel CaMKIIα-Shank3 Interaction

Tyler L. Perfitt, Xiaohan Wang, Matthew T. Dickerson…·Roger J. Colbran

This study identifies a novel interaction between Shank3 and CaMKIIα that is essential for calcium channel signaling to the nucleus and gene expression changes during learning.

54 citationstop 4%

mouseexperimental assay

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NeuroscienceCell ReportsPeer-reviewedNov 1, 2017

Amyloid accumulation drives proteome-wide alterations in mouse models of Alzheimers disease like pathology

Jeffrey N. Savas, Yizhi Wang, Laura A. DeNardo…·John R. Yates

The paper identifies proteome-wide changes in mouse models of Alzheimer's disease, highlighting ApoE dysregulation and synaptic protein loss.

71 citationstop 3%

mouseproteomics

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